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Critical research priorities include elucidating mechanisms for CRC-specific glycolytic blockade that spares normal cell viability, coupled with the development of multi-target regimens co-inhibiting EMP and complementary pathways to optimize therapeutic indices

Similarly, macrophage polarization exemplifies regulated immunometabolism: pro-inflammatory M1 macrophages exhibit high glycolytic activity to support iNOS-mediated NO production, whereas anti-inflammatory M2 macrophages depend on oxidative phosphorylation for tissue repair functions (58, 59)

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