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The mechanism likely involves copper-dependent signaling and the activation of transition metal-sensitive transcription factors (Pickart & Margolina, 2018)
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SFN mitigates TNF-a and IL-22-induced activation of inflammatory pathways and proliferation in HaCaT cells IL-22 is known to bind to the IL-22 receptor on keratinocytes, induce STAT3 phosphorylation, and promote keratinocyte proliferation [25, 26]
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