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Cell Metab 15:764777

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Because it escapes binding to these inhibitory proteins, IGF-1 LR3 remains free in experimental environments to interact continuously with the IGF-1 receptor, resulting in an extended biological half-life and vastly increased potency compared to standard IGF-1

Although the EAM mouse model is frequently used in myocarditis research because of its similarities with the pathological features and immune responses observed in human myocarditis ( 5 Conclusion The study combines animal experiments, network pharmacology, and molecular docking to reveal, for the first time, the beneficial effects of LC on energy metabolism remodeling in myocarditis, while preliminarily identifying the underlying molecular mechanisms